Fingerprint Fingerprint is based on mining the text of the persons scientific documents to create an index of weighted terms, which defines the key subjects of each individual researcher.

  • 8 Similar Profiles
Sirolimus Medicine & Life Sciences
Phosphorylation Medicine & Life Sciences
Autophagy Medicine & Life Sciences
Proteins Medicine & Life Sciences
Tuberous Sclerosis Medicine & Life Sciences
Neoplasms Medicine & Life Sciences
Phosphotransferases Medicine & Life Sciences
Intercellular Signaling Peptides and Proteins Medicine & Life Sciences

Network Recent external collaboration on country level. Dive into details by clicking on the dots.

Grants 2008 2020

Research Output 1999 2016

Aberrant mTORC1 activation kills tubular cells by inactivating miR148b-3p

Inoki, K. Dec 1 2016 In : Kidney International. 90, 6, p. 1146-1148 3 p.

Research output: Contribution to journalComment/debate

Cell Death
Endoplasmic Reticulum Stress
Tumor Necrosis Factor-alpha
1 Citations
Epithelial Cells
Kidney Diseases
5 Citations

Growth factor signaling to mTORC1 by amino acid-laden macropinosomes

Yoshida, S., Pacitto, R., Yao, Y., Inoki, K. & Swanson, J. A. Oct 12 2015 In : Journal of Cell Biology. 211, 1, p. 159-172 14 p.

Research output: Contribution to journalArticle

Intercellular Signaling Peptides and Proteins
Amino Acids
Growth Factor Receptors
19 Citations
Ribosomal Protein S6 Kinases
14 Citations

GSK3β-Dependent inhibition of AMPK potentiates activation of neutrophils and macrophages and enhances severity of acute lung injury

Park, D. W., Jiang, S., Liu, Y., Siegal, G. P., Inoki, K., Abraham, E. & Zmijewski, J. W. Nov 15 2014 In : American Journal of Physiology - Lung Cellular and Molecular Physiology. 307, 10, p. L735-L745

Research output: Contribution to journalArticle

Neutrophil Activation
AMP-Activated Protein Kinases
Macrophage Activation
Acute Lung Injury